suppressor cells造句

• CD8 + T suppressor cells predominate in patients with no protective immunity ( visceral leishmaniasis patients ).
• Myeloid-derived suppressor cells ( MDSCs ) are a heterogenous population of cells of chronic inflammation and cancer.
• Part of that defense response involves cells called CD8 lymphocytes _ the killer cells and suppressor cells of the immune system.
• Immune tolerance is an important means by which growing myeloid derived suppressor cells ( MDSCs ), which also induce peripheral tolerance.
• Once this happens, anti-HIV antibodies can bind to and kill most of the host's T suppressor cell population.
• This, Cohn said, suggested  that the suppressor cells produce a factor which, in effect, turns off the macrophage.
• Once the leishmania infection has been eliminated and leishmania antigens are gone, CD8 + T suppressor cells down-regulate CD4 + T helper cells.
• These findings suggest that antigen stimulation of CD4 + helper T cells results in production of cytokines that kill or down regulate CD8 + T suppressor cells.
• Isolation of cytokines that inhibit and kill CD8 + T suppressor cells might be useful in treating diseases that involve immune suppression such as leishmaniasis, AIDS, and certain cancers.
• Hence in this model, the onset of AIDS is primarily an auto-immune reaction triggered by the cross-reaction of anti-HIV antibodies with T suppressor cells.
• Following an infection with HIV that manages to establish itself, there is a complex interaction between the HIV virus, the T helper cells that it infects, and T suppressor cells.
• These three quasispecies apply selective pressure on one another and co-evolve in such a way that the viral epitopes eventually come to mimick the V regions of the main population of T suppressor cells.
• Additionally, a controversial new study shows that ultraviolet light encourages suppressor cells in the immune system to stifle the activity of disease-fighting cells and can degrade some nutrients in the blood, such as beta carotene.
• His research has also focused on monocytic-myeloid derived suppressor cells and polymorphonuclear-myeloid derived suppressor cells and what impact they might have on cancer therapy, since myeloid derived suppressor cells negatively regulate anti-tumor activity.
• His research has also focused on monocytic-myeloid derived suppressor cells and polymorphonuclear-myeloid derived suppressor cells and what impact they might have on cancer therapy, since myeloid derived suppressor cells negatively regulate anti-tumor activity.
• His research has also focused on monocytic-myeloid derived suppressor cells and polymorphonuclear-myeloid derived suppressor cells and what impact they might have on cancer therapy, since myeloid derived suppressor cells negatively regulate anti-tumor activity.
• The suppressor cells are not affected by glucosteroid response-modifying factor, so the effective setpoint for the immune cells may be even higher than the setpoint for physiological processes ( reflecting leukocyte redistribution to lymph nodes, bone marrow, and skin ).
• Some experiments we've been doing in the lab with cells of other diseases indicate that either or both may be possible, but we need to know a lot more about the bacteria and the immune cells before we can manipulate them clinically . . . . What we can learn about these suppressor cells . . . is also of great interest because there's a fair amount of evidence in many animal models that suppressor cells modify the ability of a host to fight off tumors, and that if you can destroy the suppressor cells, you'll allow normal body defenses to come back and destroy the tumor . 
• Some experiments we've been doing in the lab with cells of other diseases indicate that either or both may be possible, but we need to know a lot more about the bacteria and the immune cells before we can manipulate them clinically . . . . What we can learn about these suppressor cells . . . is also of great interest because there's a fair amount of evidence in many animal models that suppressor cells modify the ability of a host to fight off tumors, and that if you can destroy the suppressor cells, you'll allow normal body defenses to come back and destroy the tumor . 
• Some experiments we've been doing in the lab with cells of other diseases indicate that either or both may be possible, but we need to know a lot more about the bacteria and the immune cells before we can manipulate them clinically . . . . What we can learn about these suppressor cells . . . is also of great interest because there's a fair amount of evidence in many animal models that suppressor cells modify the ability of a host to fight off tumors, and that if you can destroy the suppressor cells, you'll allow normal body defenses to come back and destroy the tumor .